Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.
Sci. Signal., 25 October 2011
Vol. 4, Issue 196, p. eg9
Focus Issue: Recruiting Players for a Game of ERK
Nancy R. Gough1*
1 Editor of Science Signaling, American Association for the Advancement of Science, 1200 New York Avenue, N.W., Washington, DC 20005, USA.
The extracellular signal–regulated kinase (ERK) pathway is one of the superfamily of mitogen-activated protein kinase pathways. Signals transmitted by this kinase cascade activate a pair of related proteins, ERK1 and ERK2. Research published in Science Signaling shows that, despite the wealth of knowledge about this pathway, previously unknown functions continue to be discovered and additional components in this pathway continue to be identified. Thus, we are continuing to learn the plays and recruit the players in the game that is ERK signaling.
Nancy R. Gough (29 January 2013) Sci. Signal.6 (260), ec28.
[DOI: 10.1126/scisignal.2004002] |Abstract »
Adam A. Friedman, George Tucker, Rohit Singh, Dong Yan, Arunachalam Vinayagam, Yanhui Hu, Richard Binari, Pengyu Hong, Xiaoyun Sun, Maura Porto, Svetlana Pacifico, Thilakam Murali, Russell L. Finley, Jr., John M. Asara, Bonnie Berger, and Norbert Perrimon (25 October 2011) Sci. Signal.4 (196), rs10.
[DOI: 10.1126/scisignal.2002029] |Editor's Summary »|Abstract »|Full Text »|PDF »|Supplementary Materials »
THIS ARTICLE HAS BEEN CITED BY OTHER ARTICLES:
A Novel Mechanism Involving Four-and-a-half LIM Domain Protein-1 and Extracellular Signal-regulated Kinase-2 Regulates Titin Phosphorylation and Mechanics.
A. Raskin, S. Lange, K. Banares, R. C. Lyon, A. Zieseniss, L. K. Lee, K. G. Yamazaki, H. L. Granzier, C. C. Gregorio, A. D. McCulloch, et al. (2012)
J. Biol. Chem.
|Abstract »|Full Text »|PDF »