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Sci. Signal., 10 March 2009
[DOI: 10.1126/scisignal.2000162]

Supplementary Materials for:

Rac1 Is a Critical Mediator of Endothelium-Derived Neurotrophic Activity

Naoki Sawada, Hyung-Hwan Kim, Michael A. Moskowitz, James K. Liao*

*To whom correspondence should be addressed. E-mail: jliao{at}rics.bwh.harvard.edu

This PDF file includes:

  • Materials and Methods
  • Supplementary Text
  • Fig. S1. CD31 labeling of Rac1+/– and Rac1+/+ ECs.
  • Fig. S2. Rac1+/– ECs show retarded proliferation, migration, and capillary formation.
  • Fig. S3. CD31 labeling of MBECs.
  • Table S1. Representative gene sets enriched in Rac1+/+ mouse ECs.
  • Table S2. Representative gene sets enriched in Rac1+/– mouse ECs.
  • Table S3. Transcription factors differentially expressed between Rac1+/– and Rac1+/+ mouse ECs.
  • Table S4. List of differentially expressed genes between Rac1+/– and Rac1+/+ mouse ECs.

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Technical Details

Format: Adobe Acrobat PDF

Size: 2,065 KB


Citation: N. Sawada, H.-H. Kim, M. A. Moskowitz, J. K. Liao, Rac1 is a critical mediator of endothelium-derived neurotrophic activity. Sci. Signal. 2, ra10 (2009).

© 2009 American Association for the Advancement of Science


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