Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.


Sci. Signal., 18 January 2011
[DOI: 10.1126/scisignal.2001211]

Supplementary Materials for:

The Kinase SGK1 in the Endoderm and Mesoderm Promotes
Ectodermal Survival by Down-Regulating Components of the Death-Inducing Signaling Complex

Tatsuya Endo, Morioh Kusakabe,* Kazunori Sunadome, Takuya Yamamoto, Eisuke Nishida*

*To whom correspondence should be addressed. E-mail: morioh{at}lif.kyoto-u.ac.jp (M.K.); nishida{at}lif.kyoto-u.ac.jp (E.N.)

This PDF file includes:

  • Fig. S1. Characterization of xSGK1.
  • Fig. S2. Overexpression of xFADD or Xcaspase-10 causes excessive apoptosis.
  • Fig. S3. Loss of xSGK1 increases the expression of Xp53 presumably through a pathway independent of xBMP7.
  • Table S1. Primer sequences.

[Download PDF]

Technical Details

Format: Adobe Acrobat PDF

Size: 10.9 MB


Citation: T. Endo, M. Kusakabe, K. Sunadome, T. Yamamoto, E. Nishida, The Kinase SGK1 in the Endoderm and Mesoderm Promotes Ectodermal Survival by Down-Regulating Components of the Death-Inducing Signaling Complex. Sci. Signal. 4, ra2 (2011).

© 2011 American Association for the Advancement of Science


To Advertise     Find Products


Science Signaling. ISSN 1937-9145 (online), 1945-0877 (print). Pre-2008: Science's STKE. ISSN 1525-8882