Note to users. If you're seeing this message, it means that your browser cannot find this page's style/presentation instructions -- or possibly that you are using a browser that does not support current Web standards. Find out more about why this message is appearing, and what you can do to make your experience of our site the best it can be.

Subscribe

Logo for

Science 313 (5788): 767-768

Copyright © 2006 by the American Association for the Advancement of Science

IMMUNOLOGY:
When F-actin Becomes Too Much of a Good Thing

Michael L. Dustin

An excess of a protein that regulates actin assembly in white blood cells unexpectedly kills the cell by altering mitochondrial function.


The author is in the Progam in Molecular Pathogenesis, Skirball Institute, New York University Medical Center, 540 First Avenue, New York, NY 10016, USA. E-mail: dustin{at}saturn.med.nyu.edu



To Advertise     Find Products


Science Signaling. ISSN 1937-9145 (online), 1945-0877 (print). Pre-2008: Science's STKE. ISSN 1525-8882