FocusInflammation

Integrating p38α MAPK immune signals in nonimmune cells

See allHide authors and affiliations

Sci. Signal.  03 Mar 2015:
Vol. 8, Issue 366, pp. fs5
DOI: 10.1126/scisignal.aaa8398

You are currently viewing the abstract.

View Full Text

Log in to view the full text

Log in through your institution

Log in through your institution

Abstract

A study by Huang et al. in this issue of Science Signaling highlights key roles for the mitogen-activated protein kinase p38α and the phosphatase MK1 in mediating interleukin-17–dependent pathology in experimental autoimmune encephalomyelitis, a mouse model of multiple sclerosis.

View Full Text