Vol 8, Issue 391
- The G protein α subunit variant XLαs promotes inositol 1,4,5-trisphosphate signaling and mediates the renal actions of parathyroid hormone in vivo
The extra-large Gα subunit that mediates the effects of parathyroid hormone on the kidney stimulates two distinct downstream pathways.
- RelB/p52-mediated NF-κB signaling alters histone acetylation to increase the abundance of corticotropin-releasing hormone in human placenta
Glucocorticoids epigenetically induce the expression of a stress hormone gene associated with labor in human placenta cells.
Tissue damage stimulates quiescent cells to reenter the cell cycle through a mechanism that relies on Ajuba-dependent mechanical stimulation of Yorkie activity.