1Department of Health Technology, Technical University of Denmark, Kemitorvet, Building 202, 2800 Kgs Lyngby, Denmark.
2Department of Experimental Medical Science, Lund University, 22184 Lund, Sweden.
3Institute of Experimental Immunology, University of Zurich, Winterthurerstrasse 190, Zurich, Switzerland.
4Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.
5Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg University of Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
5Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg University of Mainz, Obere Zahlbacher Str. 67, Mainz 55131, Germany.
By John Rizk, Joseph Kaplinsky, Rasmus Agerholm, Darshana Kadekar, Fredrik Ivars, William W. Agace, W. Wei-Lynn Wong, Matthew J. Szucs, Samuel A. Myers, Steven A. Carr, Ari Waisman, Vasileios Bekiaris
Science Signaling
By promoting noncanonical NF-κB signaling, SMAC mimetics can reprogram CD4+ T helper cell differentiation and curb autoimmunity.
By John Rizk, Joseph Kaplinsky, Rasmus Agerholm, Darshana Kadekar, Fredrik Ivars, William W. Agace, W. Wei-Lynn Wong, Matthew J. Szucs, Samuel A. Myers, Steven A. Carr, Ari Waisman, Vasileios Bekiaris
Science Signaling
By promoting noncanonical NF-κB signaling, SMAC mimetics can reprogram CD4+ T helper cell differentiation and curb autoimmunity.