Research ArticleNeuroscience

Rac1 Is a Critical Mediator of Endothelium-Derived Neurotrophic Activity

See allHide authors and affiliations

Science Signaling  10 Mar 2009:
Vol. 2, Issue 61, pp. ra10
DOI: 10.1126/scisignal.2000162

eLetters is an online forum for ongoing peer review. Submission of eLetters are open to all. Please read our Terms of Service before submitting your own eLetter.

Compose eLetter

Plain text

  • Plain text
    No HTML tags allowed.
  • Web page addresses and e-mail addresses turn into links automatically.
  • Lines and paragraphs break automatically.
Author Information
First or given name, e.g. 'Peter'.
Your last, or family, name, e.g. 'MacMoody'.
Your email address, e.g.
Your role and/or occupation, e.g. 'Orthopedic Surgeon'.
Your organization or institution (if applicable), e.g. 'Royal Free Hospital'.
Statement of Competing Interests

This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.

Vertical Tabs

  • ACC Award

    Science Signaling congratulates first author Naoki Sawada for winning a 2009 American College of Cardiology Young Investigator Award for this research on the regulation of endothelial cell neurotrophic activity and the role of endothelial Rac1 in brain injury.

    Competing Interests: None declared.

Stay Connected to Science Signaling