ReviewCell death

The Role of the Kinases RIP1 and RIP3 in TNF-Induced Necrosis

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Science Signaling  30 Mar 2010:
Vol. 3, Issue 115, pp. re4
DOI: 10.1126/scisignal.3115re4

Article Information

vol. 3 no. 115 re4

DOI: 
https://doi.org/10.1126/scisignal.3115re4
PubMed: 
20354226
Published By: 
Science Signaling
Print ISSN: 
1945-0877
Online ISSN: 
1937-9145
History: 
  • .
Copyright & Usage: 
Copyright © 2010, American Association for the Advancement of Science

Author Information

  1. Peter Vandenabeele1,2,*,
  2. Wim Declercq1,2,
  3. Franky Van Herreweghe1,2, and
  4. Tom Vanden Berghe1,2
  1. 1Molecular Signaling and Cell Death Unit, Department for Molecular Biomedical Research, VIB, 9052 Zwijnaarde, Belgium.
  2. 2Department of Biomedical Molecular Biology, Ghent University, 9052 Zwijnaarde, Belgium.
  1. ↵*Corresponding author. E-mail, Peter.Vandenabeele{at}dmbr.UGent.be

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  • The Role of the Kinases RIP1 and RIP3 in TNF-Induced Necrosis

    By Peter Vandenabeele, Wim Declercq, Franky Van Herreweghe, Tom Vanden Berghe

    Science Signaling : re4

    Programmed necrosis in response to TNF requires the activity of two serine-threonine kinases.

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