PerspectiveImmunology

CD69: An Unexpected Regulator of TH17 Cell–Driven Inflammatory Responses

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Science Signaling  22 Mar 2011:
Vol. 4, Issue 165, pp. pe14
DOI: 10.1126/scisignal.2001825

Article Information

vol. 4 no. 165 pe14

DOI: 
https://doi.org/10.1126/scisignal.2001825
PubMed: 
21427408
Published By: 
Science Signaling
Print ISSN: 
1945-0877
Online ISSN: 
1937-9145
History: 
  • .
Copyright & Usage: 
Copyright © 2011, American Association for the Advancement of Science

Author Information

  1. Pilar Martín1,* and
  2. Francisco Sánchez-Madrid1,2,*
  1. 1Department of Vascular Biology and Inflammation, Fundación Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC), 28029 Madrid, Spain.
  2. 2Servicio de Inmunología, Hospital de La Princesa, Universidad Autónoma de Madrid, 28006 Madrid, Spain.
  1. ↵*Corresponding authors. E-mail, pmartinf{at}cnic.es (P.M.); fsanchez.hlpr{at}salud.madrid.org (F.S.-M.)

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  • CD69: An Unexpected Regulator of TH17 Cell–Driven Inflammatory Responses

    By Pilar Martín, Francisco Sánchez-Madrid

    Science Signaling : pe14

    CD69 deficiency promotes differentiation of inflammatory TH17 cells and exacerbates autoimmune diseases in mice.

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