Research ArticleCell Biology
Protein kinase A–dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor 1
- John W. Bullen1,2,
- Irina Tchernyshyov3,
- Ronald J. Holewinski3,
- Lauren DeVine4,
- Fan Wu3,
- Vidya Venkatraman3,
- David L. Kass3,
- Robert N. Cole4,
- Jennifer Van Eyk3, and
- Gregg L. Semenza1,2,3,4,5,6,7,*
- 1Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 2McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 3Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 4Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 5Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 6Department of Pediatrics, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- 7Department of Radiation Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
- ↵*Corresponding author. Email: gsemenza{at}jhmi.edu
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Science Signaling 31 May 2016:
Vol. 9, Issue 430, pp. ra56
DOI: 10.1126/scisignal.aaf0583