Supplementary Materials

Supplementary Materials for:

RelB/p52-mediated NF-κB signaling alters histone acetylation to increase the abundance of corticotropin-releasing hormone in human placenta

Valeria Di Stefano, Bingbing Wang,* Nataliya Parobchak, Natalie Roche, Todd Rosen*

*Corresponding author. E-mail: wangbi{at}rwjms.rutgers.edu (B.W.); rosentj{at}rwjms.rutgers.edu (T.R.)

This PDF file includes:

  • Fig. S1. Both acetylation and deacetylation of H3K9 are involved in the expression of COX-2 in term human placenta.
  • Fig. S2. Silencing of CBP or HDAC1 attenuates dexamethasone-induced up-regulation of CRH or COX-2 in term human placenta.
  • Fig. S3. p300 is not associated with the CRH promoter in term human placenta.
  • Fig. S4. The RelB/p52/CBP/HDAC1 complex is not associated with the CRH promoter in midtrimester placentas.

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Citation: V. Di Stefano, B. Wang, N. Parobchak, N. Roche, T. Rosen, RelB/p52-mediated NF-κB signaling alters histone acetylation to increase the abundance of corticotropin-releasing hormone in human placenta. Sci. Signal. 8, ra85 (2015).

© 2015 American Association for the Advancement of Science