Supplementary Materials

Supplementary Materials for:

The NF-κB subunit RelB controls p100 processing by competing with the kinases NIK and IKK1 for binding to p100

Amanda J. Fusco, Anup Mazumder, Vivien Ya-Fan Wang, Zhihua Tao, Carl Ware, Gourisankar Ghosh*

*Corresponding author. Email: gghosh{at}ucsd.edu

This PDF file includes:

  • Fig. S1. Aberrant subcellular distributions of p52 and p100 in Relb−/− cells.
  • Fig. S2. NIK and RelB compete for binding to p100.
  • Fig. S3. RelB stimulates kappaBsome formation.
  • Fig. S4. RelB protects p100 from complete degradation.
  • Fig. S5. RelB, NIK:IKK1, and p100 form a multiprotein complex.

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Citation: A. J. Fusco, A. Mazumder, V. Y.-F. Wang, Z. Tao, C. Ware, G. Ghosh, The NF-κB subunit RelB controls p100 processing by competing with the kinases NIK and IKK1 for binding to p100m. Sci. Signal. 9, ra96 (2016).

© 2016 American Association for the Advancement of Science