Supplementary Materials

Supplementary Materials for:

Wdpcp promotes epicardial EMT and epicardium-derived cell migration to facilitate coronary artery remodeling

Xiangyang Liu, Ye Wang, Feng Liu, Min Zhang, Hejie Song, Bin Zhou, Cecilia W. Lo, Shilu Tong, Zhenlei Hu,* Zhen Zhang*

*Corresponding author. Email: zhenzhang{at}sjtu.edu.cn (Z.Z.); 13564677103{at}163.com (Z.H.)

This PDF file includes:

  • Fig. S1. Expression of Wdpcp in embryonic heart.
  • Fig. S2. Ciliary defects but normal transcriptional response to Shh signaling in Wdpcp mutant hearts.
  • Fig. S3. Normal formation of the intramyocardial endothelial plexus.
  • Fig. S4. Normal egression of coronary endothelial cells from the sinus venosus.
  • Fig. S5. Proliferative and apoptotic status of coronary endothelial cells in E13.5 hearts.
  • Fig. S6. Normal coronary vessel formation in E18.5 embryos with myocardium-specific deletion of Wdpcp.
  • Fig. S7. Wdpcp knockdown recapitulates the gel infiltration defect of Wdpcp mutant-derived EPDCs.
  • Table S1. List of qPCR primers used in the study.

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