Supplementary Materials

This PDF file includes:

  • Fig. S1. Effect of MeHg (100 ppm) on body weight and protein amounts.
  • Fig. S2. Subneurotoxic dose of MeHg promotes induction of maladaptive gene expression and remodeling after pressure overload.
  • Fig. S3. Changes in the abundance of mitochondrial fusion- and fission-related proteins.
  • Fig. S4. Effect of MeHg exposure on cardiomyocyte apoptosis.
  • Fig. S5. MeHg exposure minimally affects Drp1 phosphorylation in the heart.
  • Fig. S6. Depolysulfidation of recombinant Drp1 by MeHg.
  • Fig. S7. Hypotonic stress–induced apoptosis of MeHg-exposed NRCMs.
  • Fig. S8. Uncropped Western blots for Figs. 2 to 4.
  • Fig. S9. Uncropped Western blots for Figs. 5 and 6.
  • Fig. S10. Uncropped Western blots for fig. S1.
  • Fig. S11. Uncropped Western blots for figs. S2 and S3.
  • Fig. S12. Uncropped Western blots for figs. S4 to S7.
  • Table S1. Echocardiographic parameters of mice with 100-ppm MeHg exposure at 1 or 2 weeks.
  • Table S2. Echocardiographic parameters of TAC-operated mice with MeHg exposure.
  • Table S3. Millar catheter analysis of TAC-operated mice given MeHg and NaHS.

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